Hip Osteoarthritis: Symptoms, Signs and Diagnosis
What hip osteoarthritis actually feels like, where the pain comes from, what a surgeon finds on examination, and what your X-rays mean — explained clearly for patients and their families.
Hip osteoarthritis is the second most common form of osteoarthritis after the knee, and one of the leading causes of pain and disability in adults over the age of 50.1 Despite how prevalent it is, patients frequently arrive in clinic having lived with significant symptoms for years, unsure whether what they are experiencing is truly their hip, uncertain about where to seek help, and often surprised to learn how much can be done — both before and after a decision about surgery.
This article explains what hip osteoarthritis feels like from a patient’s perspective, what a surgeon looks for on examination, and what your X-rays actually mean.
Where does hip osteoarthritis hurt?
The most characteristic symptom of hip osteoarthritis is groin pain — a deep, aching discomfort felt in the front of the hip crease, which may radiate down the inner thigh towards the knee.1,2 Many patients are surprised by this because they expect hip pain to be felt on the outer side of the hip or buttock. In fact pain felt directly over the lateral hip — the bony prominence you can feel on the outer thigh — more commonly comes from the bursa or tendons around the hip, not from the joint itself.
That said, hip osteoarthritis can also present as:2,3
- Anterior thigh pain — aching in the front of the thigh, sometimes confused with muscle pain
- Knee pain — referred pain from the hip can travel all the way to the knee via the obturator nerve, and it is not uncommon for patients to present to clinic convinced their knee is the problem when it is actually their hip
- Buttock pain — deep posterior discomfort, sometimes confused with sciatica or lumbar spine disease
- Lateral hip pain — less typical, but can occur, particularly in more advanced disease
This variability in pain location is one reason why hip osteoarthritis is sometimes misdiagnosed or delayed. If you have been told you have knee pain, a back problem, or a muscle strain and treatment has not helped, it is worth asking whether your hip has been properly assessed.
What does the pain feel like?
The pain of hip osteoarthritis tends to have a fairly characteristic pattern, though it varies between patients and changes as the disease progresses.2,4
In the early stages, pain is typically:
- Activity-related — brought on by walking, climbing stairs, getting in and out of a car, or putting on shoes and socks
- Relieved by rest — sitting down or lying still eases it
- Associated with start-up stiffness — a familiar stiffness and aching after sitting for a period that eases after a few minutes of walking, sometimes called the “gelling phenomenon”
- Worse at the end of the day after activity
As the disease progresses, the pattern changes:
- Pain occurs with less activity — short walks that were previously manageable become difficult
- Rest pain develops — discomfort that is present even when sitting still
- Night pain emerges — waking in the night with hip discomfort, often when turning over in bed, is a sign of more advanced disease and a significant indicator that the condition is having a major impact on quality of life
- Morning stiffness may increase, though in osteoarthritis it typically lasts less than 30 minutes — stiffness lasting longer than this should raise the possibility of inflammatory arthritis5
What else might you notice?
Beyond pain, patients with hip osteoarthritis commonly report:2,3
- Reduced range of movement — difficulty crossing legs, cutting toenails, getting in and out of a low car, or performing rotational movements
- A limp — an antalgic gait (limping to offload the painful hip) or, in more advanced cases, a Trendelenburg gait caused by weakness of the hip abductor muscles
- Leg length discrepancy — in severe hip OA, the femoral head can migrate upward within the joint, causing a functional shortening of the affected leg
- Clicking or clunking — though this is less specific and can occur in normal hips
- Giving way — particularly on uneven ground, related to muscle weakness and loss of proprioception
- Impact on daily activities — difficulty with stairs, prolonged walking, housework, sport, and in some patients, basic self-care
What does a surgeon find on examination?
When a surgeon or clinician examines your hip, they are looking for a combination of findings that together build a picture of how your joint is functioning and how severe the disease is likely to be. The examination typically takes place with you lying on a couch and involves careful, systematic assessment of movement and strength.
The findings most strongly associated with hip osteoarthritis on examination are:6
- Reduced and painful internal rotation — internal rotation (turning the foot inward with the knee bent) is consistently the first movement to be lost in hip OA and is highly sensitive for the diagnosis. Reduced internal rotation has a positive likelihood ratio of 3.2 for radiographic OA6
- Reduced hip adduction — bringing the leg across the midline; a positive likelihood ratio of 4.26
- Groin pain on passive abduction or adduction — pain reproduced by moving the leg outward or inward has a positive likelihood ratio of 5.76
- Hip abductor weakness — weakness of the gluteal muscles, tested by resisted abduction or the Trendelenburg test, has a positive likelihood ratio of 4.56
- Posterior hip pain on squatting — the most specific single finding, with a positive likelihood ratio of 6.16
- An antalgic or Trendelenburg gait — observed as you walk into the consulting room, before any formal examination begins
- Fixed flexion deformity — assessed using the Thomas test; the inability to fully straighten the hip indicates contracture of the hip flexors, a feature of more advanced disease
A finding with a high positive likelihood ratio means that when it is present, it substantially increases the probability that osteoarthritis is the diagnosis. These examination findings are therefore not just routine — they are diagnostically meaningful.
What do your X-rays show?
Plain X-rays of the hip remain the first-line investigation for suspected hip osteoarthritis and are usually all that is needed to confirm the diagnosis and plan treatment.5 A standard assessment includes an anteroposterior (AP) view of the pelvis — showing both hips together for comparison — and a lateral view of the affected hip.
The radiographic features of hip osteoarthritis are:7,8
- Joint space narrowing — the most important finding. The apparent “space” between the femoral head and the acetabulum on X-ray represents the cartilage. As it is lost, this space narrows. Superior joint space narrowing (loss at the top of the joint) is the most common pattern in hip OA
- Osteophytes — bony spurs growing around the margins of the joint, visible as irregular projections at the edges of the femoral head and acetabulum
- Subchondral sclerosis — increased whiteness (density) in the bone immediately beneath where the cartilage has been lost, as the bone attempts to compensate for the absent cushioning
- Subchondral cysts — small fluid-filled cavities within the bone near the joint surface
- Femoral head deformity — in more advanced disease, the femoral head itself loses its spherical shape and may migrate superolaterally (upward and outward) within the socket
These features are graded using the Kellgren-Lawrence (KL) classification, the most widely used system in clinical practice:8,9
- Grade 0 — no radiographic features of OA
- Grade 1 — doubtful: possible joint space narrowing with subtle osteophyte formation
- Grade 2 — mild: definite joint space narrowing with definite osteophytes and slight sclerosis
- Grade 3 — moderate: marked joint space narrowing, multiple osteophytes, sclerosis, cyst formation, and early femoral head deformity
- Grade 4 — severe: near-complete or complete loss of joint space, large osteophytes, gross deformity of the femoral head and acetabulum
Established, clinically significant OA is generally defined as KL grade 2 or above. However — and this is important — the grade of your X-ray does not determine whether you need surgery. Surgeons operate on patients, not X-rays. A grade 4 X-ray in a patient who is coping well and has not tried conservative measures is not an automatic indication for surgery. A grade 2 X-ray in a patient whose quality of life is severely affected and who has exhausted all non-surgical options may well be.
When might further imaging be needed?
In most cases, X-rays are all that is required. However, MRI or CT scanning may be requested in specific circumstances:
- MRI — if the diagnosis is uncertain, if inflammatory arthritis is suspected, if there is concern about avascular necrosis (bone death due to loss of blood supply), or if soft tissue pathology such as a labral tear needs assessment
- CT — primarily used for surgical planning, particularly in complex or revision cases, to assess bone stock and anatomy in three dimensions
- Bone scan or SPECT-CT — occasionally used when the source of pain is genuinely unclear and multiple joints or regions are involved
How is hip OA distinguished from other causes of hip pain?
Not all hip pain is osteoarthritis. The most important conditions to consider in the differential diagnosis are:3,5
- Greater trochanteric pain syndrome (GTPS) — formerly called trochanteric bursitis, this presents as lateral hip pain that is point tender over the greater trochanter. It is often confused with hip OA but is a distinct condition affecting the tendons and bursa on the outer hip
- Lumbar spine disease — L3/4 nerve root pathology can refer pain to the groin and anterior thigh, mimicking hip OA. The “hip-spine syndrome” — concurrent hip and spine disease — is common and can make diagnosis challenging
- Inflammatory arthritis — rheumatoid arthritis, psoriatic arthritis, and reactive arthritis can all affect the hip. Features suggesting inflammatory arthritis include bilateral joint involvement, prolonged morning stiffness (over 30 minutes), constitutional symptoms, and elevated inflammatory markers (CRP, ESR)
- Avascular necrosis (AVN) — death of bone tissue due to disrupted blood supply, associated with steroid use, alcohol excess, sickle cell disease, and previous hip fracture. Can mimic OA on early X-rays but has a distinct appearance and often more rapid progression
- Femoroacetabular impingement (FAI) — abnormal contact between the femoral head and acetabulum, typically presenting in younger patients with activity-related groin pain and restricted internal rotation
- Referred pain from the knee or spine — pain that seems to come from the hip but originates elsewhere
An experienced clinician can usually distinguish these conditions on the basis of history and examination, with targeted investigations where needed. Getting the diagnosis right is the essential first step — because the treatment for each of these conditions is different.
- The most characteristic symptom of hip OA is groin pain — not lateral hip pain.
- Hip pain can also refer to the thigh and knee, which is why it is sometimes initially misdiagnosed.
- Hip OA pain is typically activity-related in early stages, progressing to rest pain and night pain as the disease advances.
- Night pain is an important indicator of severity.
- Loss of internal rotation is the earliest and most consistent examination finding in hip OA, and is highly diagnostically significant.
- X-ray grade (Kellgren-Lawrence 0–4) describes the structural severity of the disease — but the grade alone does not determine whether surgery is needed.
- Symptoms and quality of life are equally important.
- Not all hip pain is osteoarthritis. Greater trochanteric pain syndrome, lumbar spine disease, AVN, and inflammatory arthritis all cause hip pain and require different treatment.
- If knee pain has not responded to knee-directed treatment, ask whether your hip has been properly assessed — referred pain from the hip to the knee is common.
Likelihood Ratios in Clinical Examination The Metcalfe et al systematic review (JAMA 2019) formally quantified the diagnostic accuracy of clinical findings for hip OA using likelihood ratios. A positive likelihood ratio (LR+) greater than 5 is generally considered to substantially increase the probability of a diagnosis; an LR+ greater than 10 is considered highly significant. The finding of squat causing posterior hip pain (LR+ 6.1) and groin pain on passive abduction or adduction (LR+ 5.7) are therefore among the most diagnostically powerful clinical tests in musculoskeletal medicine. Conversely, being under 60 years of age has a negative likelihood ratio (LR-) of 0.11 — meaning that in a younger patient, the pretest probability of hip OA is substantially lower and alternative diagnoses should be more actively considered. The Hip-Spine Syndrome The concept of hip-spine syndrome — coined by Offierski and MacNab in 1983 — describes the clinical challenge of concurrent hip and lumbar spine pathology producing overlapping symptoms. It is estimated that up to 20% of patients presenting with hip OA have coexisting lumbar spine disease. Distinguishing the relative contribution of each is critical because operating on the wrong joint first leads to persistent symptoms and patient dissatisfaction. Diagnostic selective injections — blocking the hip joint under image guidance and assessing symptom response — can be a valuable tool in ambiguous cases. Radiographic Grading — Limitations of Kellgren-Lawrence The KL classification, while widely used, has well-documented limitations. It was originally designed for epidemiological research rather than individual clinical decision-making, and interobserver reliability can be modest, particularly for grades 1 and 2. The modified Croft Score for the hip places equal weighting on femoral osteophytes and joint space narrowing, and may offer advantages in early-stage disease. For routine clinical practice, KL remains the standard, but surgeons and radiologists should be aware of its limitations when making treatment decisions. Key References Metcalfe D, Perry DC, Claireaux HA, et al. Does this patient have hip osteoarthritis? The rational clinical examination systematic review. JAMA. 2019;322(23):2323–2333. doi:10.1001/jama.2019.19413 Aresti N, Kassam J, Nicholas N, Achan P. Hip osteoarthritis. BMJ. 2016;354:i3405. doi:10.1136/bmj.i3405 Lane NE. We challenged the Kellgren and Lawrence radiographic scoring method and came up with some interesting epidemiology for osteoarthritis of the hip. HSS J. 2023;19(4):402–406. doi:10.1177/15563316231192514
All articles on this site are evidence-based and referenced to peer-reviewed literature, national guidelines, and registry data. References are in Vancouver format as used in medical journals.
1. Palazzo C, Ravaud JF, Papelard A, Ravaud P, Poiraudeau S. The burden of musculoskeletal conditions. PLoS One. 2014;9(3):e90633. Epidemiology
doi:10.1371/journal.pone.0090633 · PMID: 24599100
2. Lespasio MJ, Sultan AA, Piuzzi NS, Khlopas A, Husni ME, Muschler GF, et al. Hip osteoarthritis: a primer. Perm J. 2018;22:17–084. Review
doi:10.7812/TPP/17-084 · PMID: 29493488
3. Aresti N, Kassam J, Nicholas N, Achan P. Hip osteoarthritis. BMJ. 2016;354:i3405. Clinical Review
doi:10.1136/bmj.i3405 · PMID: 27440180
4. Hunter DJ, Bierma-Zeinstra S. Osteoarthritis. Lancet. 2019;393(10182):1745–1759. Seminal Review
doi:10.1016/S0140-6736(19)30417-9 · PMID: 31034380
5. National Institute for Health and Care Excellence. Osteoarthritis in over 16s: diagnosis and management. NICE Guideline NG226. London: NICE; October 2022. NICE Guideline
Available at: nice.org.uk/guidance/ng226
6. Metcalfe D, Perry DC, Claireaux HA, Appelbe D, Chesser TJS, Costa ML, et al. Does this patient have hip osteoarthritis?: the rational clinical examination systematic review. JAMA. 2019;322(23):2323–2333. Systematic Review
doi:10.1001/jama.2019.19413 · PMID: 31846019
7. Kellgren JH, Lawrence JS. Radiological assessment of osteo-arthrosis. Ann Rheum Dis. 1957;16(4):494–502. Original Article
doi:10.1136/ard.16.4.494 · PMID: 13498604
8. Lane NE. We challenged the Kellgren and Lawrence radiographic scoring method and came up with some interesting epidemiology for osteoarthritis of the hip. HSS J. 2023;19(4):402–406. Review
doi:10.1177/15563316231192514 · PMID: 37937093
9. van der Kroef M, Vincken KL, Bartels LW, Custers RJ, Viergever MA, Tuijthof GJ, et al. Kellgren/Lawrence grading in cohort studies: methodological update and implications illustrated using data from a Dutch hip and knee cohort. Arthritis Care Res. 2022;74(10):1639–1647. Methodology
doi:10.1002/acr.24756 · PMID: 33650774
10. Offierski CM, MacNab I. Hip-spine syndrome. Spine. 1983;8(3):316–321. Original Article
doi:10.1097/00007632-198304000-00014 · PMID: 6224892
11. Valdes AM, Spector TD. Genetic epidemiology of hip and knee osteoarthritis. Nat Rev Rheumatol. 2011;7(1):23–32. Review
doi:10.1038/nrrheum.2010.191 · PMID: 21079645
- The most characteristic symptom of hip OA is groin pain — not lateral hip pain.
- Hip pain can also refer to the thigh and knee, which is why it is sometimes initially misdiagnosed.
- Hip OA pain is typically activity-related in early stages, progressing to rest pain and night pain as the disease advances.
- Night pain is an important indicator of severity.
- Loss of internal rotation is the earliest and most consistent examination finding in hip OA, and is highly diagnostically significant.
- X-ray grade (Kellgren-Lawrence 0–4) describes the structural severity of the disease — but the grade alone does not determine whether surgery is needed.
- Symptoms and quality of life are equally important.
- Not all hip pain is osteoarthritis. Greater trochanteric pain syndrome, lumbar spine disease, AVN, and inflammatory arthritis all cause hip pain and require different treatment.
- If knee pain has not responded to knee-directed treatment, ask whether your hip has been properly assessed — referred pain from the hip to the knee is common.
Likelihood Ratios in Clinical Examination The Metcalfe et al systematic review (JAMA 2019) formally quantified the diagnostic accuracy of clinical findings for hip OA using likelihood ratios. A positive likelihood ratio (LR+) greater than 5 is generally considered to substantially increase the probability of a diagnosis; an LR+ greater than 10 is considered highly significant. The finding of squat causing posterior hip pain (LR+ 6.1) and groin pain on passive abduction or adduction (LR+ 5.7) are therefore among the most diagnostically powerful clinical tests in musculoskeletal medicine. Conversely, being under 60 years of age has a negative likelihood ratio (LR-) of 0.11 — meaning that in a younger patient, the pretest probability of hip OA is substantially lower and alternative diagnoses should be more actively considered. The Hip-Spine Syndrome The concept of hip-spine syndrome — coined by Offierski and MacNab in 1983 — describes the clinical challenge of concurrent hip and lumbar spine pathology producing overlapping symptoms. It is estimated that up to 20% of patients presenting with hip OA have coexisting lumbar spine disease. Distinguishing the relative contribution of each is critical because operating on the wrong joint first leads to persistent symptoms and patient dissatisfaction. Diagnostic selective injections — blocking the hip joint under image guidance and assessing symptom response — can be a valuable tool in ambiguous cases. Radiographic Grading — Limitations of Kellgren-Lawrence The KL classification, while widely used, has well-documented limitations. It was originally designed for epidemiological research rather than individual clinical decision-making, and interobserver reliability can be modest, particularly for grades 1 and 2. The modified Croft Score for the hip places equal weighting on femoral osteophytes and joint space narrowing, and may offer advantages in early-stage disease. For routine clinical practice, KL remains the standard, but surgeons and radiologists should be aware of its limitations when making treatment decisions. Key References Metcalfe D, Perry DC, Claireaux HA, et al. Does this patient have hip osteoarthritis? The rational clinical examination systematic review. JAMA. 2019;322(23):2323–2333. doi:10.1001/jama.2019.19413 Aresti N, Kassam J, Nicholas N, Achan P. Hip osteoarthritis. BMJ. 2016;354:i3405. doi:10.1136/bmj.i3405 Lane NE. We challenged the Kellgren and Lawrence radiographic scoring method and came up with some interesting epidemiology for osteoarthritis of the hip. HSS J. 2023;19(4):402–406. doi:10.1177/15563316231192514
All articles on this site are evidence-based and referenced to peer-reviewed literature, national guidelines, and registry data. References are in Vancouver format as used in medical journals.
1. Palazzo C, Ravaud JF, Papelard A, Ravaud P, Poiraudeau S. The burden of musculoskeletal conditions. PLoS One. 2014;9(3):e90633. Epidemiology
doi:10.1371/journal.pone.0090633 · PMID: 24599100
2. Lespasio MJ, Sultan AA, Piuzzi NS, Khlopas A, Husni ME, Muschler GF, et al. Hip osteoarthritis: a primer. Perm J. 2018;22:17–084. Review
doi:10.7812/TPP/17-084 · PMID: 29493488
3. Aresti N, Kassam J, Nicholas N, Achan P. Hip osteoarthritis. BMJ. 2016;354:i3405. Clinical Review
doi:10.1136/bmj.i3405 · PMID: 27440180
4. Hunter DJ, Bierma-Zeinstra S. Osteoarthritis. Lancet. 2019;393(10182):1745–1759. Seminal Review
doi:10.1016/S0140-6736(19)30417-9 · PMID: 31034380
5. National Institute for Health and Care Excellence. Osteoarthritis in over 16s: diagnosis and management. NICE Guideline NG226. London: NICE; October 2022. NICE Guideline
Available at: nice.org.uk/guidance/ng226
6. Metcalfe D, Perry DC, Claireaux HA, Appelbe D, Chesser TJS, Costa ML, et al. Does this patient have hip osteoarthritis?: the rational clinical examination systematic review. JAMA. 2019;322(23):2323–2333. Systematic Review
doi:10.1001/jama.2019.19413 · PMID: 31846019
7. Kellgren JH, Lawrence JS. Radiological assessment of osteo-arthrosis. Ann Rheum Dis. 1957;16(4):494–502. Original Article
doi:10.1136/ard.16.4.494 · PMID: 13498604
8. Lane NE. We challenged the Kellgren and Lawrence radiographic scoring method and came up with some interesting epidemiology for osteoarthritis of the hip. HSS J. 2023;19(4):402–406. Review
doi:10.1177/15563316231192514 · PMID: 37937093
9. van der Kroef M, Vincken KL, Bartels LW, Custers RJ, Viergever MA, Tuijthof GJ, et al. Kellgren/Lawrence grading in cohort studies: methodological update and implications illustrated using data from a Dutch hip and knee cohort. Arthritis Care Res. 2022;74(10):1639–1647. Methodology
doi:10.1002/acr.24756 · PMID: 33650774
10. Offierski CM, MacNab I. Hip-spine syndrome. Spine. 1983;8(3):316–321. Original Article
doi:10.1097/00007632-198304000-00014 · PMID: 6224892
11. Valdes AM, Spector TD. Genetic epidemiology of hip and knee osteoarthritis. Nat Rev Rheumatol. 2011;7(1):23–32. Review
doi:10.1038/nrrheum.2010.191 · PMID: 21079645